Sports neurogenic disease is a group of patients with unclear selective infringement of spinal cord cells, brain stem neurons, cortical cone cells and cone bundle beams. The incidence rate is about 1 to 3 / 100,000 per year, the prevalence is 4 to 8/100 per year. Since most patients died in 3 to 5 years after symptoms, the prevalence rate and incidence of the disease were relatively close. The Mnd cause is unclear, generally considered to be due to age, caused by genetic susceptibility individuals to adverse to adverse environments, that is, genetic factors and environmental factors lead to the occurrence of motor neuronal disease.
Symptoms of sports neuron disease
1, mixed types usually in hand muscle weakness, shrinking as the first symptom, generally affecting the opposite side after the beginning, with the development of the course of disease, the lower motion neuron mixed damage symptoms, called muscle atrophy lateral rigidosis. In the late disease, the whole body muscles are slightly shrinking, so that the head is not enough, the breathing is difficult, the bed is not affordable. The disease is in the age of 40 to 60, about 5 ~ 10% have a history of family genetic, and the disease progresses is very slow.
2, the lower moving neurons typically use the small muscle weakness and muscle of the hand, and can wave and one or both sides, or the opposite side will be transmitted from one side. Due to the atrophy of the smashing muscle, the palm of the palm, the bone muscle, etc., is a clawed. The muscle atrophy is expanded, gradually infringes the forearm, upper arm and shoulder strap. The muscle bundle is common, which can be limited to some muscle or widely existed, tapping with hand, easily induce.
3, the performance of the upper sports neurons is the limb weakness, tightening, and the movement is not working. The symptoms start from double lower limbs, and the biphypertensive are later, and the following limbs are heavy. The weak limbs is weak, the muscle tension is increased, the step is difficult, and the scissors, the tendon reflection is hyperthyroidism, and the pathological reflection is positive. If the sick is denied and the bilateral cortex dry, the symptoms of pseudo ball paralysis are present, and the pronunciation, swallowing obstacles, hypervide, and the like.
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